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Cranial Nerve Testing

Unlabelled image of cranial nerves

Unlabelled diagram of cranial nerves. Image by by jlcampbell104 is marked under CC0 1.0

Overview

Labelled diagram of cranial nerves. Image by by jlcampbell104 is marked under CC0 1.0

Olfactory I

Quick method – ask the patient if:

If they say yes to any of these questions, ask if they have a blocked nose or a cold at the moment, to save you doing unnecessary further investigation! If they say ‘yes’ ask if they think this is the source of their reduced sense of smell.
Also ask about taster here, in case you forget later! Sense of taste is likely to be impaired when the sense of smell is impaired!

 
Proper Method – odours on cotton wool is a bit old fashioned. Usually now clinical testing involves scratchcards with a scratch and sniff panel. These cards are usually from the USA, and have flavours like ‘Blureberry pie’

Optic II

Assess visual acuity

Start by asking a few basic questions:

Then, ask the patient to take off their glasses, and have a good look at and around the eyes (e.g. swelling, erythema, ptosis), before beginning your formal examinations:

Visual fields 

Sit at arm’s length away from the patient and perform the following tests:

Pupillary Light Reflex

Assess pupillary light reflex for ipsilateral and contralateral eye. The optic nerve only carries afferent fibres of the pupillary light reflex.

Miosis – means ‘constricted pupil’!
Causes:

Mydriasis – means ‘dilated pupil’!
Causes
Fixed oval pupil – likely to be glaucoma
Grossly irregular pupil – most likely cause is adhesion that are the result of iritis.
The following external link has an interactive ‘Eye Simulator’ to test reflexes:
http://cim.ucdavis.edu/EyeRelease/Interface/TopFrame.htm

Accommodation reflex

Fundoscopy

The optic disc – is it normal colour? Normal size? Normal shape?

The vessels – do they form the normal arc? Are they tortuous? Can you see any haemorrhage?
The retina – any abnormalities (e.g. cotton wool spots, exudates)
Cloudy appearance? – if so, likely to be cataracts.
Ask the patient to look up, down, left and right so you can get a view of all the areas of the retina.
Ask the patient to look directly at the light – so you can see the macula. Don’t do this for too long!
Finally, move away slightly, or refocus the ophthalmoscope to look in the vitreous for floaters, and to look at the iris and sclera.

Diabetic eye changes:

Background retinopathy

Proliferative retinopathy

 
Retinis Pigmentosa

Oculomotor III

Ask the patient to follow your finger. Have your finger about30cm away from the patient’s face, and move it in an H shape to test all the eye movements.
Ask the patient if they experience double vision at any time

Oculomotor nerve palsy – Complete / incomplete, Medical/surgical

Trochlear IV

Suberior obliquethis causes a lot of confusion in medical students. This muscle will point the eye downwards, when the eye is looking medially!
If it was to ever act on the eye alone (when no other muscle are acting on the eye) then yes, it might make the eye look laterally. However, in clinical practice, this never happens!

Ask the patient if they ever experience double vision. Ask them to look directly downwards, then far to the left, and far to the right.

Trochlear Nerve palsy –this can be difficult to detect as it is subtle. It also often presents with other palsies / lesions.

Trigeminal V

Test their sensory function. Touch patient with cotton wool in the 3 regions (ophthalmic, maxillary, mandibular) on both sides of the face.
The ophthalmic (touch on forehead), the maxillary (touch on cheek bones), and the mandibular (touch on chin). If they can’t feel in an of these regions, then they might have a lesion in the corresponding cranial nerve.
Be wary in the mandibular region – that along the region of the mandibular bone, sensory function is actually due to C2, so it is best practice to touch just laterally to the mouth.
Test the integrity of the motor function of the mandibular (V3) branch of the trigeminal nerve:

Test the corneal reflex – touch their cornea with a cotton wool bud. The patient should close their eyes. This test the ophthalmic branch of the trigeminal nerve and the zygomatic branch of the facial nerve.

Abducens VI

Lateral rectus – if its not working, you’ll have trouble looking laterally. Ask the patient if they have experienced double vision. Ask them to look directly far to the left and directly far to the right.

Abducens palsy – patient is unable to abduct the eye

Facial VII

Vestibulocochlear VIII

 
There are two types of deafness:
We can detect which type of deafness is present by using 2 different tests:

Rinné’s Test

DO NOT HIT THE TUNING FORK ON ANYTHING! Pluck it, or hit in on your hand.
The healthy normal ear is more sensitive to air conducted sound than to bone conducted sound.This test involves places a vibrating tuning fork on the mastoid process behind your ear, then when the sound is no longer heard, bring it round to your ear, and yu should still be able to hear the sound.

Weber’s Test

You should place a vibrating tuning fork in the midline of your skull, and it should make an equal sound in both ears. If it doesn’t then there is a problem. This test is useless on its won though, because it doesn’t tell you what the problem is! Just that there is a problem.
If there is conductive deafness in one ear, the sound will be louder in that ear because background noise is being blocked out and so you ficus more on the sound you can hear, so it is louder. However, an ear may also appear louder, if the other one is relatively quieter due to sensory-neural deafness in the quieter ear.
With the help of Rinne’s test you can identify if there is conduection deafness or not in the louder ear, and thus identify whether the difference in loudness of ears is due to conduction of sensory-neural deafness.
Sensory-Neural Deafness

This can be complete 100% deafness, or any amount between normal hearing and complete deafness. In the vast majority of cases it is caused by degeneration of the hair cells in the organ of corti. This occurs normally in old age and results in loss of high frequencies (above 1 kHz). The higher the frequency, the greater the loss. This type of deafness is known as Presbycusis. Noise trauma can also cause deafness. Typically this has a hearing loss in the range of 4kHz (like the graph on the previous page). Both of these conditions will normally present in both ears. Sensorineural deafness in only one ear is possible due to brain/vestibulocochlear neve damage and should be investigated with an MRI scan.

Conduction Deafness
This can be caused by a perforated tympanic membrane, or by a middle ear infection, such as otitis media. Otitis media is a painful inflammation of the middle ear usually caused by viral infection. It is more common in children due to their shorter eustachian tubes, but it normally resolves itself as the child grows. Grommets may be inserted to aid ventilation of the middle ear (which is the normal function of the eustachain tube. In otitis media the sound conduction deafness is caused by accumulation of fluid in the middle ear, impairing movement of the mechanical apparatus of the middle ear, and thus reducing the strength of conduction to the cochlea. Otitis media is common in colds and flu, but will usually settle within 72 hours. It may last for several years in children.

Glossopharyngeal IX

Vagus X

Accessory XI

Test the integrity of the sternocleidomastoid and trapezius. Ask them to rotate their head against resistance (SCM), and to shrug their shoulders against resistance (trapezius).

Hypo​glossoal XII

Asses tongue movements. It will deviate to the weaker side. Ask if they have ever noticed any trouble talking because their tongue won’t do what they want it to.
Always explain what you’re doing! Say what you would expect to see if it was damaged.

References

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