Contents
Introduction
- The Achilles tendon is the largest tendon in the body. Tendinopathy and rupture can affect both athletes and those with sedentary lifestyles
- Acute injury (i.e. rupture) to the Achilles tendon usually occurs from sudden acceleration or deceleration. This is different to Achilles tendinopathy – which is often a chronic overuse condition
- Achilles tendon rupture occurs in athletes in 80% of cases. It is more common in those who have had previous Achilles tendon problems
- Achilles tendon rupture can be partial or complete
- Complete tears are usually managed surgically, but in older sedentary patients may be managed conservatively. Surgical management reduces the risk of re-rupture, but has a higher acute complication rate
Aetiology
- Usually in athletes
- Most commonly in sports that involve a quick acceleration – e.g. sprinting, tennis, basketball, football
- Typically aged 30-50
- M > F
- May also occurs as a result of a fall with forces dorsiflexion
- More common in chronic Achilles tendinopathy
- Drugs
- Corticocosteroids
- Fluoroquinolones – e.g. ciprofloxacin
- Avoid use of both of these medications together!
- Pre-existing conditions
- Rheumatoid arthritis or other inflammatory rheumatological disorders
Pathology
- The Achilles tendon connects the gastrocnemius and soles muscles to the posterior of the calcaneus
- It begins at the mid calf
- It has slightly unusual anatomy – there is not true synovial sheath and instead it is surrounded by a sheath called the paratenon.This is a highly vascular structure and it allows for a sliding action of the tendon inside the paratenon
- The blood supply is least around the mid tendon – defined as the region 2-6cm proximal to the insertion. This is the area most likely to rupture
Presentation
- Sudden onset Achilles tendon pain. Initially sharp pain, often settles within hours to days to more of a dull ache
- A loud “snap” or “bang” is sometimes heard when the tendon ruptures!
- Some patients report feeling as though they were hit on the back of the tendon
- Usually occurs with a pushing-off motion – in contact to ankle sprain, which is often due to impact on landing
- Inability to stand on tip toe on affected side
Examination
- Often localised swelling
- Altered gait
- Inability to stand on tip toes
- Reduced plantar flexion
- Simmond’s Triad. Ask the patient to lay prone on the couch, with their feet dangling off the end of the bed. Compare both sides. Check:
- Altered “angle of dangle” – in Achilles tendon rupture the affect foot will naturally “dangle” in a more dorsiflexed position than the unaffected side
- Calf squeeze (aka Thomson’s calf squeeze test) – in a normal, intact Achilles tendon, a calf squeeze should cause plantar flexion of the ankle. In Achilles tendon rupture, there will be minimal or no plantar flexion
- Palpable notch or gap in the Achilles tendon at this site of the rupture
Investigation
- Often a clinical diagnosis
- USS (usually first line) or MRI can help to differentiate a partial from a complete tear, or to narrow down differentials
Management
- Non-weight bearing – immediately
- Urgent referral to orthopaedics – surgery is usually required
- Surgical repairs reduces the risk of subsequent recurrent rupture, but also carries risks – such as wound infection
- Following surgery, rehabilitation plan is often similar to conservative management outlined below
- 80% of athletes return to sport
- Athletes may not be able to resume full activity for up to 1 year
- There is often some loss of function – even small amount of functional loss can have a significant impact for athletes
- Conservative management is an option for older more sedentary patients – but decision should be made by orthopaedic specialist
- Plaster cast for 2 weeks
- Can be changed to immobilising boot from 2 weeks
- Weight bear as pain allows from 4-6 weeks
- Physiotherapy involvement from the start
- Partial tears are usually managed conservatively, with an immobilisation boot from the start, and earlier weight bearing
Complications
- Achilles tendon scarring and contracture – especially if there is not early mobilising and range of movement exercises – best direct by physiotherapist
- Re-rupture
- 3-5% after surgical treatment
- 8-12% with conservative management
- DVT